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Deficiency of znT8 promotes adiposity and metabolic dysfunction by increasing peripheral serotonin production

Studying the virus/host interactome and associated pathology will contribute to a better understanding of the largest immune compartment in the body, the GI tract, and its role in the immunological acceptance of gut microbes

Introduction Chronic oxidative stress is commonly considered a major contributor to the pathogenesis of metabolic diseases, such as obesity-linked type 2 diabetes, metabolic dysfunction-associated steatohepatitis (MASH), and associated complications like diabetic kidney and heart disease

It emerges that these senescent cells can be eradicated in mice using a drug that interferes with the activity of the protein FOXO4