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Chronic hyperglycemia in T2DM has been reported to induce significant alterations in DNA methylation, subsequently resulting in increased risks of cardiovascular dysfunction [111, 114,115,116] by promoting oxidative stress, atherosclerosis, endothelial dysfunction and dysregulated lipid metabolism

It helps recycle oxidized glutathione back to its active form, effectively boosting the body's antioxidant capacity

Abstract Tumor heterogeneity is a fundamental driver of therapeutic resistance across solid malignancies, arising from genetic, epigenetic, phenotypic, spatial, temporal, and microenvironmental diversity

After evaporation of the solvent and reduction of pressure, n -hexane fraction (HLHE, 3.2 g) and residue were obtained

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