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EAAT2/G93A mSOD1 double Tg mice showed moderate amelioration of the ALS-like phenotype with a statistically significant (14 days) delay in grip strength decline and loss of motor neurons as well as a reduction in other events including caspase-3 activation and SOD1 aggregation but not in the onset of paralysis, body weight decline or an extended life span when compared with monotransgenic G93A mSOD1 littermates (Guo et al., 2003)

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Gastroenterology 149 , 17841793 (2015)