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Our results suggest that ablation of the three sites results in a loss of function that constrains RyR2 activity, losing part of the physiological regulation of SR Ca 2+ leak during adrenergic stimulation

These findings confirmed a certain degree of similarity between Gclc -deficient FoB and Gclc -sufficient MZB, which was further substantiated by comparing differentially expressed genes between the two groups Gclc fl/fl MZB vs

One of the key proposed contributors to these conditions is oxidative stress, which is thought to impair energy production in muscles, leading to the characteristic fatigue observed in these patients

*Correspondence: Viktria Jeney [email protected] This article was submitted to Inflammation, a section of the journal Frontiers in Immunology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Lockhart et al., 2023)