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Mitochondrial ros-induced lysosomal dysfunction impairs autophagic flux and contributes to M1 macrophage polarization in a diabetic condition

10.1074/jbc.M201062200 39 JohanssonA.-S.MannervikB

Cong, J

10.1016/j.dld.2014.02.010 138

In L02 cells overexpressed with FABP1, the total intracellular FABP1 proteins gradually declined in response to treatment with CHX, a protein synthesis inhibitor [35], whereas overexpression of GSTA1 led to an acceleration of the decline with a shortening of the half-life of FABP1 (Figure 7E)