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Furthermore, the CAR gene structure itself can impose a metabolic burden, leading to higher internal ROS levels in CAR-NK cells compared to their normal counterparts [242]

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The applicant further opined that, although not explicitly stated, the paper presumably calculated the sample size based on the primary endpoint of stent removal, not to detect any clinical differences and, while Johnson et al

In summary, while GBMs unique metabolic characteristics confer a significant survival advantage to tumor cells, they also expose a key vulnerability

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