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In: Arias IM, Boyer JL, Chisari FV

Wilson Disease and low ceruloplasmin Ceruloplasmin A multi-copper blood ferroxidase involved in iron and copper homeostasis and inflammation

(2) Mechanistic superiority: Dual inhibition of TBK1 and IKK in eWAT restores AMPK-Thr172 phosphorylation, enhancing mitochondrial biogenesis (via UCP1 upregulation), catecholamine sensitivity (via cAMP upregulation and cAMP-mediated -adrenergic signal transduction) [201, 202]