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Mutations in the major histocompatibility complex class I-like transmembrane protein, specifically the H63D mutation in the HFE gene, and mutations in the hemojuvelin (HJV) gene, both associated with systemic iron overload, have been linked to elevated hypertension risk [240, 241]

1 Introduction -1 antitrypsin (A1AT) (Alpha-1 Proteinase Inhibitor, 1-Pi) belongs to the serine protease inhibitor superfamily (SERPIN)

1a) 9 , which under normal physiological conditions is predominantly expressed in astrocytes within the brain and macrophages 10,11

This reactivity underlies cysteines central role in enzyme catalysis, where it often serves as the nucleophilic active site residue in numerous enzyme families

Further, there is a need to elucidate the dynamic interactions between different iron sources, gut microbiota composition, and host physiological responses