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72 Mutant p53 3KR , which is defective for the conventional p53 functions, still be able to repress SLC7A11 and induce ferroptosis upon ROS-induced stress

SAC treatment markedly reduced ROS levels, restored GSH levels, and attenuated MDA accumulation, demonstrating its efficacy in alleviating oxidative stress and lipid peroxidation in UC mice

In view of the known toxicity of sucrose to mammalian cells, a variant might be to use an isoosmotic 4.5% (w/v) solution of Nycodenz, which has the same density

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