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Jilin: Changchun University of Traditional Chinese Medicine

Interestingly, SLC38A9 is important for the arginine-dependent export of leucine from the lysosomal lumen, highlighting the crosstalk between AA that activate mTORC1 (Wyant et al., 2017)

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In addition to APAP-induced direct hepatotoxicity, the intracellular signaling mechanisms of APAP-induced liver injury (AILI) including metabolic activation, mitochondrial oxidant stress and proinflammatory response further affect progression and severity of AILI