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Apigenin attenuates pulmonary hypertension by inducing mitochondria-dependent apoptosis of PASMCs via inhibiting the hypoxia inducible factor 1KV1

It emphasizes the core molecular structures of the above pathways, their dynamic interactions, and their key roles in alveolar epithelial apoptosis, immune imbalance, and pulmonary fibrosis, providing a theoretical basis and new therapeutic strategies for targeted inhibition of the inflammatory storm and restoration of redox homeostasis (Figure 2)

Everyones goals, body, and treatment schedules are different

The development of systems approaches together with pioneering omic technologies and computational tools will lead the way for future translational systems medicine