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In addition, the likelihood of developing NASH increases with the severity of obesity.11,36 There is a universal association between NASH an insulin resistance regardless of body mass index, suggesting that insulin resistance is a central factor in the pathogenesis of NASH.38 The elevated expression of TNF-a in the liver seen in NAFLD can represent a link between the development of insulin resistance and hepatic steatosis

Gene expression profiling of oxidative stress on atrial fibrillation in humans

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an increased rate of resistance (in ~ 30% patients) due to inconsistent pharmacokinetics, genetic variability in TKI binding sites and associated toxicities nilotinib was introduced as a second line TKI with a separate binding site

Additionally, an increase in the abundances of Proteobacteria and Streptococcus species has been linked to the development of rheumatoid arthritis (Imhann et al., 2016