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Copper, a vital trace element, is indispensable for the maintenance of physiological functioning, particularly in the cardiac system

Disrupting the clock in male mice via adipocyte-specific genetic deletion or high-fat-diet feeding reduces complex I respiration in adipocytes, leading to suppression of the peroxisome proliferator-activated receptor and insulin signalling pathways

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Although thought to be improbable, alteration of bacterial membranes has been shown as a mechanism of resistance (Epand et al., 1858

The ATP7B gene provides instructions for making this protein, a copper-transporting ATPase type 2, which functions as a pump that removes copper from the body via biliary excretion