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The findings highlight pathways such as fibroblast-to-myofibroblast transition (FMT), redox balance regulation, lysosomal dysfunction, electron transport chain (ETC) dysfunction, autophagy, and calcium dysregulation as potential contributors to hEDS pathogenesis

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At the Nasseri Clinic of Arthritic and Rheumatic Diseases, we specialize in treating gout, a form of inflammatory arthritis that causes sudden, severe pain, swelling, and redness in joints, often starting at the big toe

Nature 426 , 787 (2003)

T cell subset-specific susceptibility to aging