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Other pathways that have been implicated in hepatocyte injury and the development of NASH, although rather broadly, include cholesterol accumulation in hepatocytes (96) and a tangled web involving activation of apoptotic pathways with ER stress and abnormal unfolded protein response (97), as well as defects in autophagy (98)

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During this process, a physical therapist stimulates the affected muscle with a very fine needle

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Arabidopsis sulfate transporter AtSULTR1;1 (Shibagaki and Grossman, 2004) was used as a positive control