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Additionally, inflammatory mediators in the bone microenvironment amplify local RANKL/RANK signaling, linking systemic and localized immune responses to bone pathology (44)

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IGF-1R can initiate the activation of the PI3K/AKT/mTOR signaling and Ras/Raf/MEK/MAPK pathways resulting in the activation of multiple transcription factors such as ELK-1, CREB and AP-1 to modulate cell proliferation, survival, differentiation, motility, invasion and angiogenesis