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While the dithiol mechanism for EcGrx1 and EcRNR or EcPR can be explained by specific protein-protein interactions (Fig

Other areas involved in cognition such as the amygdala, prefrontal cortex can also be affected (Block et al., 2005

Western blotting showed that both SLC7A11 and GPX4, which activate inhibitory signals, were expressed at higher levels in the GSH-treated group than in the control group, similar to the expression pattern of the adipogenesis-related marker PPAR (Fig