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Funding The authors declare that no funds, grants, or financial support were received during the preparation of this manuscript

The reactive metabolites generated after drug biotransformation can covalently bind to cellular proteins, lipids, and nucleic acids, leading to triggering immune responses and cellular damage (Fig

Martnez-Leal et al., 2020)

Reduce the damaging impact of fatty liver illness on cells A lack of antioxidants might worsen hepatic cell death and result in fatty liver

Many ip genes had been identified in T-even phage genomes by detection of the signal sequence, but only one had been functionally characterized: Ip1 in phage T4 is an inhibitor of a Type IV restriction endonuclease (RE) known as GmrSD, which specifically cleaves glucosyl-hydroxymethylcytosine (g-hmC)-modified DNA typical of T-even phages (T2, T4, T6) 29