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These findings suggest that neuronal cells in AD disease upregulate ferritin and downregulate FPN expression, increasing iron intake and reducing iron excretion

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These developments are consistent with the evolving understanding of the gastrointestinal tract as a key endocrine organ that regulates appetite, glucose homeostasis, energy expenditure, and gastrointestinal motility through a complex network of hormonal signals [20]

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No statistically significant increases in chromosomal aberrations were observed in CHO cells at any tested concentration