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These effects lead to hippocampus-dependent cognitive loss, including difficulties with spatial learning and memory, as well as neurodegeneration, inflammation, and synaptic malfunction

In addition, intracellular oxidative damage can activate apoptosis via the C-Jun N-terminal kinase (JNK) pathway, a MAPk that activates p53

BPC-157 is also credited with being able to repair the damage caused by IBD (inflammatory bowel disease) in a few days when taken orally

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Table 4 summarizes the studies on natural bioactive compounds and dietary interventions in DKD, along with their corresponding levels of evidence