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These findings suggest that lactylation resulting from elevated lactate levels during the I/R phase is associated with key cellular processes, including energy metabolism, neuronal injury, and repair

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Demonstrating the hormetic benefit of this response in a variety of brain cells, overexpression of PGC-1 protected against cell death induced by H 2 O 2 or paraquat treatment, and this occurred in conjunction with changes in gene expression similar to those observed with the 10T1/2 cells [76]

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