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These include altered choline metabolism, changes in neurotransmitter metabolism, defective DNA methylation with consequent poly (ADP-ribose) polymerase (PARP) overactivation and PARP-controlled cell death, excitotoxicity, up-regulation of re-entry to the cell division cycle, hypomethylation of myelin lipids and myelin basic protein, white matter changes, and the formation of homocysteine thiolactone and subsequent protein homocysteinylation (89)

doi: 10.3389/fendo.2019.00346 Received 30 January 2019 Accepted 13 May 2019 Published 07 June 2019 Volume 10 - 2019 Edited by John Lui Yovich, Pivet Medical Center, Australia Reviewed by Syeda Nureena Zaidi, Pivet Medical Center, Australia

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& Lee, S.-H