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NF-B and ER stress in advanced atherosclerosis During the last decades, people have been trying to understand the pathophysiology of atherosclerosis, though the precise mechanisms underlying plaque destabilization still remain unclear

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A change in GSH and GSSG levels are important when assessing toxicological responses and an indicator of oxidative stress, potentially leading to apoptosis or cell death

Hydrogen sulfide perturbs mitochondrial bioenergetics and triggers metabolic reprogramming in colon cells

Additionally, ATP depletion was effectively mitigated, and ROS production was reduced, with the nanoparticle system demonstrating lower ROS levels compared to untreated cells