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Second, we hypothesized that chronic, exogenous activation of this excitatory pathway could reverse the crossed cerebellar diaschisis, and possibly even atrophic changes, that occur following contralateral cortical ischemia and contribute to loss of function [90, 94, 120]

In 2000, a research study was carried out in obese Zucker rats where the AOD 9604 peptide was given daily for 19 consecutive days