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This approach, coupled with high-throughput technologies, allows for the rapid identification of candidate genes involved in epileptogenesis (Liu et al., 2023)

In addition, when MK4 was incubated with rhFSP1 and NADH, MK4-hydroquinone was generated (Fig

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Blocking Pathological Opening of mPTP and Ensuring Neuronal Survival The abnormal opening of the mitochondrial permeability transition pore (mPTP) is a critical trigger for neuronal apoptosis in neurodegenerative diseases: Under pathological conditions, stimuli such as ischemia and oxidative stress can lead to excessive opening of mPTP, causing collapse of the mitochondrial membrane potential and swelling or rupture of mitochondria