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This accumulation may be due to factors such as mutations in -catenin, deficiencies in the -catenin destruction complex, most frequently by mutations in structurally disordered regions of APC , overexpression of Wnt ligands, loss of inhibitors and/or decreased activity of regulatory pathways (such as the Wnt/calcium pathway)

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ALDH1A1 drives prostate cancer metastases and radioresistance by interplay with AR- and RAR-dependent transcription