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In particular, the ability of estrogen to suppress postnatal thymocyte development and T cell production has been well-documented

Excess copper can promote ferritin depletion and disrupt iron metabolism, thereby sensitizing cardiomyocytes to ferroptosis, a process that synergistically aggravates I/R injury [67]

reported that the selective vulnerability of SN DA neurons in PD is causally related to the activation of ATP-sensitive potassium (K ATP ) channels in these neurons [179]