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moreover, the degree of Cu deficiency in mutant SOD1 aggregates is proportional to the clinical severity of ALS

While the cysteine decomposition is coupled with mitochondrial bioenergetics in the cells with intact KEAP1-NRF2 system, in NRF2-addicted cancer cells, most of the available cysteine appears to flow into the anabolic pathway, namely, glutathione synthesis and leaves the catabolic pathway in mitochondria, which is consistent with the observation that mitochondrial respiration is limited in NRF2-addicted cancer cells (Sayin et al

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The discussion on BPC 157 can be explored in more detail in episodes of the RELATED QUESTIONS