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LPS targets host guanylate-binding proteins to the bacterial outer membrane for non-canonical inflammasome activation

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Chromatin remodeling becomes progressively dysregulated with age, resulting in altered nucleosome positioning, histone mark redistribution, and accessibility changes that influence local methylation patterns

Impaired catabolism of branched-chain amino acidsmodulated by pathogenic variants in BCKDHA and loss-of-function alleles in PPM1Kcompromises tissue-selective disposal of these metabolites, thereby fostering insulin resistance and accelerating the progression of type 2 diabetes mellitus (30)

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