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Together, these findings indicated that K-Hcy-modified extracellular HMGB1/2 promotes SASP by interfering with SLIT2-ROBO4-mediated anti-inflammatory signaling, thereby exacerbating endothelial paracrine senescence

Mechanistically, gut microbiota-derived SCFAs exert their functions by activating trans-membrane G-protein-coupled receptors (GPCRs) of GPR41 and GPR43 or inhibiting histone acetylation (HDAC) to directly enter host cells (Moniri and Farah, 2021)

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This indicates that longer exposure to hydrogen, or different concentrations may have a more beneficial effect, or that hydrogen may only be effective in mild cases of brain injury [46]

Much less is known about the -Glutamyl di- peptides