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Based on these findings, the possible mechanisms of action of AGE on the process of neuroinflammation in A-induced AD have been proposed (51)

This dysregulation of CAT and subsequent ROS accumulation contribute to vascular injury, which in turn plays a pivotal role in the development of carotid plaque formation and bleeding, thereby contributing to the progression of AS [130]

Solubility and stability were as described

IL-27 even widely influences TIGIT, LAG-3, CTLA-4, and PD-1 expression by activating STAT1 and B lymphocyte-induced maturation protein 1 (BLIMP1) [258]

Data availability The data that support this study are available from the corresponding author upon reasonable request