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Senescent mitochondria not only produce more ROS due to inefficient electron transport but also exhibit impaired iron-sulfur cluster biogenesis, leading to improper iron handling and accumulation within the mitochondria, thus creating a potent internal source of oxidative stress (106,107)

Due to its defined peptide sequence and metal-binding properties, it is utilized in laboratory systems examining peptide stability, coordination chemistry, and molecular response mechanisms under strictly controlled scientific conditions

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When the mitochondrial membrane is enriched in cholesterol, it decreases the activity of the GSH transporter system [57]