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It regulates numerous genes associated with ferroptosis, including those involved in GSH synthesis, NADPH regeneration, and iron metabolism (such as iron storage and export, as well as heme synthesis and degradation)

Ideal for follow-up testing to monitor detoxification progress

The findings support further investigation but do not prove that all products described as liposomal are equivalent

Moreover, excess iron could promote the formation of reactive oxygen species (ROS), which can lead to oxidative stress and DNA damage, with cytokine-regulated inflammatory pathways and a central contribution from innate immune cells in the tumor microenvironment (360, 361)

6.2 Innovation in clinical trial design Traditional PD trials are constrained by: Slow progression of disease markers