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Zhang DD, Hannink M (2003) Distinct cysteine residues in Keap1 are required for Keap1-dependent ubiquitination of Nrf2 and for stabilization of Nrf2 by chemopreventive agents and oxidative stress

Happle and Kster were unable to demonstrate a bimodal distribution of phenotypes with clearly unaffected and clearly affected individuals as is usually seen in autosomal dominant disorders (51)

GSH is the most important antioxidant and free radical scavenger that is found to be decreased in the brains of people with a wide range of mental and neurological illnesses from schizophrenia[iv] to dementia[v] [vi]

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We conclude that the main function of endogenous GSH is not to limit inflammation but to fine-tune the innate immune response to infection