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Although the detailed mechanisms of the different behaviors of Gpx4 depending on cell types remain unknown, we suggest that ferroptosis, triggered by Gpx4 downregulation and excessive LPs, may be the critical cell death pathway that underlies the susceptibility of cardiomyocytes to DOX-induced injuries

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Recent 2025 research has taken this further, developing dimeric copper peptide formulations incorporated into smart hydrogels that respond to the wound environment and provide controlled release of the active compound [3]

the resulting nitrogen mustard induces interstrand and intrastrand DNA crosslinks, which are responsible for its cytotoxic effects (Barnes et al

Derakhshani B, Jafary H, Zanjani BM et al (2020) Combined QTL mapping and RNA-Seq profiling reveals candidate genes associated with cadmium tolerance in barley