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In this manuscript, we propose that an integrative neurogenomics workflowcombining multi-omic phenotypeoriented whole-exome sequencing, biologically-oriented common variant burden profiling, biochemical markers, functional metagenomic data, and multidisciplinary clinical interpretationcan generate testable, personalized diagnostic and therapeutic hypotheses in neurodegenerative disorders

PMID 12192052

In vitro, FSP1-deficient BMDMs displayed lowered mRNA levels of proinflammatory cytokines and increased anti-inflammatory cytokines upon lipopolysaccharide (LPS)/interferon- (IFN-) stimulation (Fig

68 In the cerebellum, 8-oxo-dG was negatively associated with GSH/GSSG in the combined case and control cohort (Figure 3)