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Mitigation of depleted uranium-induced mitochondrial damage by ethylmalonic encephalopathy 1 protein via modulation of hydrogen sulfide and glutathione pathways

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The C57BI/6 mouse was fed with - glucan and observed that they were evolved to have a diabetic metabolic phenotype despite possessing the same genetic determinants, suggesting that the alteration in the gut microbiota population may be a significant factor in the development of diabetic metabolic phenotype (258)

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