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*Correspondence: Mirjana Macvanin, [email protected] This article was submitted to Cardiovascular Endocrinology, a section of the journal Frontiers in Endocrinology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

PKC also participates broadly in RTK-driven migration/proliferation and in stress-network formation involving MAPKs, STAT3, and NF-B [255,256,257,258]

O.HuppertzH.-J.et al

HSA contains 35 cysteine residues, 34 of which form disulfide bridges and only one free sulfhydryl group exists, located at position 34 7,8,9,10

Symptomatic oHCM is a genetic condition where patients have thickened heart muscles and reduced blood flow from the left side of the heart to the rest of the body causing shortness of breath, fatigue, and potentially life-threatening events