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doi: 10.1016/j.critrevonc.2023.104068 172 GalvoFHFTraldiMCCArajoRSSStefanoJTD'AlbuquerqueLACOliveiraCP

In neonatal rat cardiomyocytes subjected to doxorubicin-induced stress, GKT137831 at 5 M significantly improved cell viability, reduced ROS production, and decreased apoptosis by lowering pro-apoptotic markers (PARP, CC3, BAX) while increasing anti-apoptotic Bcl-2 expression [183]