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Thyroid hormones act both on liver, regulating hepatic lipid metabolism and hepatosteatosis, and in adipose cells, increasing energy expenditure and enhancing lipolysis (160, 161)

30 min pretreatment, 24 h AO exposure) dose-dependently inhibits the release of proinflammatory mediators (TNF-, IL-1, NO) and the expression of proinflammatory proteins (iNOS, COX-2) induced by AO in BV-2 cells, suppresses AO-induced oxidative stress, and restores the antioxidant capacity of cells [5]

The amino acid transport transcriptional regulators Ahr1 and Stp2 contribute to activating inflammasome-inducing pyroptosis by hampering phagosomal acidification (258)

and c) replication hindrance, where incorporation points in DNA are blocked