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This concurrently triggers the production of highly reactive lipid aldehydes which covalently interact with electron transport chain proteins, initiating vicious cycles of increased rates of mitochondrial ROS generation (59)

Therefore, while the exact mechanisms controlling CHAC1 expression requires further study, a significant connection to the TP53 pathway is evident

These isoenzymes play a role in elimination of toxic aldehydes, which are produced during lipids peroxidation

This means that it does not have a natural model of action and may potentially cause unwanted side effects during long-term administration