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however, these results have not been reported with the use of other agents such as cyclophosphamide

Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiency

In the context of MS, the proliferation and differentiation of neural stem cells and neural precursor cells may be disrupted

Figure 9B and Supplemental Figure 14K), similarly to vehicle- and VPA-treated Kat6b +/+ mice

ATP [edit] ATP has complex concentration dependent effects on GLUD1 activity: Low [ATP] - inhibition, mediated through the GTP-binding site, since it is eliminated by H507Y