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Additionally, pinocembrin has been shown to restore cognitive function and reduce neuronal apoptosis and hippocampal inflammation in vivo, mainly by inhibiting NLRP3 inflammasome formation, reducing microglial infiltration, and enhancing BNIP3-mediated mitophagy through the JNK-ERK signaling pathway [175]

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This sequential activation

Ferroptosis is an iron-dependent lipid peroxidation-mediated cell death

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