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Figure 1 G6PD Knockdown on Infected Cells Exacerbates the Virus-Induced Oxidative Stress and Influenza Virus Replication As stated above, basal G6PD activity contributes to the regeneration of NADPH that is required for the maintenance of the cellular GSH pool (Salvemini et al., 1999)

Additionally, excess metal can be shuttled into nerve cells, provoking iron-mediated neurotoxicity

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