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These data in experimental models of AD with old 5XFAD mice could probably explain why the clinical efficacy of the monotherapeutic MEM approach is lost in moderate to severe memory deficits in advanced AD patients [37]

The compound consists of 15 amino acids in a specific sequence

The administration of the selective agonist 3-(1,1-dimethylbutyl)-1-deoxy-8-tetrahydrocannabinol (JWH-133) to rats for 9 days reduced inflammatory infiltration, - SMA expression, type I collagen storage and increased the MMP-2 expression (Muoz-Luque et al., 2008)

Both compounds have anti-inflammatory properties through different mechanisms

Include slight excess to account for dead volume in syringes