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9 Summary The available literature indicates that non-medical use of exogenous testosterone and other anabolicandrogenic steroids (AAS) is associated with a broad spectrum of multisystem adverse effects mediated by oxidative stress, mitochondrial dysfunction, endocrine disruption, and epigenetic mechanisms

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During pathological conditions such as AP, MCU becomes hyperactive, leading to excessive Ca 2+ accumulation in the mitochondrial matrix, which overwhelms the mitochondrias buffering capacity