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GPxs, which participate in the processes of antioxidant protection and detoxification, are important enzymes that directly regulate ROS levels [28]

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HMGB1 triggers an inflammatory response in macrophages through the AGER pathway, resulting in the release of TNF

Together, these findings highlight the Goldilocks role of ROS: too little fails to induce effector T cell formation and instead promotes Tregs, whereas excessive or prolonged ROS drives exhaustion and apoptosis

Glycine, cysteine, and glutamate leave the blood by transport into liver cells (depending on their concentrations) and they are also formed in the blood by the breakdown of GSH and GSSG into their component amino acids [37, 38]