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However, to date, the main molecular and cellular drivers of systemic aging of the immune system are still largely unclear

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The presence of the peptide in these pivotal locations might enable a more efficient immune response, which could be a factor in the observed scientific findings

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Functional validation using receptor-deficient models confirms that 3-adrenergic signaling is required for these effects, as disruption of this pathway attenuates peptide-induced lipolysis

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