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The resulting GABA then potentially affects the brain via the vagus nerve via the gut-brain axis or by increasing GABA levels in the blood

Together, these findings underscore the bifunctionality of cysteine in regulating protein translation besides serving as the substrate for GSH biosynthesis (Fig

Molecular Basis of G6PD Deficiency In principle, genetically determined deficiency of G6PD, like that of any other protein, might be attributed either to quantitative changes, such as mutations that affect the amount of the enzyme but not its structure, or to qualitative changes, such as mutations that affect the structure of the enzyme and hence its stability or catalytic efficiency