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A randomized controlled trial of intranasal ketamine in major depressive disorder

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Hyperglycemia and dyslipidemia stimulate NLRP3 activation via reactive oxygen species (ROS), promoting inflammatory responses and -cell pyroptosis, thereby worsening glucose metabolism dysregulation ( Oxidative stress further exacerbates diabetes pathogenesis by generating excess ROS through glucose autooxidation and the formation of advanced glycation end products (AGEs) ( 2.6 Self-immune response and genetic factors The self-immune response is the bodys immune response to its own tissues, including the autoimmune attack on pancreatic -cells, which is the main pathogenic mechanism of T1D (Velloso et al., 2013)

Limited glutamine resources in the microenvironment can inhibit mTOR activity and subsequently affect immune cell function

Do not use on children or animals